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  药店国别: 荷兰药房
产地国家: 荷兰
所属类别: 皮肤科药物->肌肤干燥
处方药:处方药
包装规格: 100克
计价单位:
   
生产厂家英文名:
Galderma
原产地英文商品名:
Calmurid (Calmuril equivalent) Cream 100g
中文参考商品译名:
Calmurid乳剂 (Calmuril equivalent)100克
原产地国家批准上市年份:
0000/00/00
英文适应病症1:
dry skin
英文适应病症2:
hyperkeratosis
临床试验期:
完成
中文适应病症参考翻译1:
皮肤干燥
中文适应病症参考翻译2:
角化过度
药品信息:
Calmurid - 糖皮质激素。抑制白细胞和组织巨噬细胞的功能。白细胞在炎症区域的迁移。削弱巨噬细胞的吞噬作用的能力和白细胞介素-1的生成。有助于溶酶体膜的稳定化,从而减少炎症的字段的蛋白水解酶的浓度。降低毛细血管的通透性,由于组胺释放。 抑制成纤维细胞和胶原形成的作用。抑制磷脂酶A2的这导致抑制前列腺素和白三烯的合成的活性。抑制环氧合酶(主要是COX- 2 ) ,释放这也有助于减少前列腺素的产生。 减少循环淋巴细胞(T -和B-细胞),单核细胞,嗜酸性粒细胞和嗜碱性粒细胞的数目,从血管床向淋巴组织的位移的结果是,能够抑制抗体的形成。 氢化可的松抑制垂体促肾上腺皮质激素和β-促脂解素的释放,但不减少循环的β -内啡肽的水平。抑制TSH和FSH的分泌。 与直接应用接收器的具有血管收缩作用。 氢化可的松对的碳水化合物,蛋白质和脂肪的代谢具有显着的剂量依赖性作用。刺激糖异生,促进氨基酸由肝脏和肾脏的捕获和增加糖异生酶的活性。中氢化可的松的肝脏通过刺激糖原合酶和合成从蛋白质代谢的产物葡萄糖的活性增加糖原沉积。增加的血糖激活胰岛素的分泌。 氢化可的松抑制葡萄糖的脂肪细胞在癫痫发作,导致活化脂肪分解。然而,由于胰岛素分泌增加是脂肪生成的刺激而导致脂肪堆积。 这种药物在淋巴和结缔组织,肌肉,脂肪组织,皮肤,骨组织的分解代谢作用;在较小程度上比盐皮质激素影响的水和电解质代谢的过程:促进钾离子和钙,延迟钠和水的身体的排泄。骨质疏松症和Itsenko - 库欣\ 's综合征是限制GCS的长期治疗的主要因素。由于分解代谢行动的结果可能会抑制儿童生长发育。 在高剂量氢化可的松可以增加脑组织的兴奋性,并有助于降低惊厥准备的门槛。它刺激的过度生产,促进消化道溃疡的发展胃中盐酸和胃蛋白酶。 供系统使用氢化可的松引起的抗炎,抗过敏,免疫抑制和抗增殖作用的治疗活性。当外部和本地应用氢化可的松的治疗活性是由于抗炎,抗过敏和antiexudative (因血管收缩效果)的动作。对于抗炎活性这个药比泼尼松龙弱4倍,在盐皮质激素活性明显优于其他GCS 。 适应症:皮肤干燥,角化过度 适应症:皮肤疾病 适应症:适应症为皮质类固醇响应dematoses的炎症表现的救济时,因继发感染,通过生物易受氯碘羟喹庆大霉素引起复杂的是抗感染剂,同时具有抗菌和anticandidal活性受感染的皮肤疾病。 Calmurid阿尔卑斯化学是糖皮质激素。 Calmurid阿尔卑斯化学抑制白细胞和组织巨噬细胞的功能。白细胞在炎症区域的迁移。削弱巨噬细胞的吞噬作用的能力和白细胞介素-1的生成。有助于溶酶体膜的稳定化,从而减少炎症的字段的蛋白水解酶的浓度。降低毛细血管的通透性,由于组胺释放。抑制成纤维细胞和胶原形成的作用。 (全身使用) :内分泌disordes (原发性或继发性肾上腺皮质功能不全)冲击反应迟钝常规治疗,如果肾上腺皮质功能不全或存在怀疑, Rheumati Ç障碍(后 - 创伤性骨关节炎,骨性关节炎的滑膜炎,类风湿性关节炎,急性和亚急性滑囊炎上髁炎。 ,急性非特异性腱鞘炎,急性痛风性关节炎,银屑病关节炎胶原性疾病(系统性红斑狼疮,急性风湿性心脏炎,系统性dermatomysitis ) ,皮肤病学疾病(天疱疮,重症多形erythma ,剥脱性皮炎,重度银屑病)的眼科疾病(过敏性和炎症性过程涉及眼,如:眼带状疱疹,虹膜炎, iridocy clitis ,脉络膜视网膜炎,角膜炎) ,胃肠道疾病(溃疡性结肠炎全身theropy )呼吸系统疾病(症状性结节病,铍中毒)血液学病症(获得[免疫性]溶血性贫血),肿瘤性疾病。 (局部使用) :各种皮肤疾病(如昆虫叮咬,毒橡树/常春藤,湿疹,皮炎,过敏,皮疹,外女性生殖器瘙痒,肛门瘙痒) 。氢化可的松可减少肿胀,瘙痒,红肿,可以发生在这些类型的条件。这种药物是一种温和的皮质类固醇。 Calmurid - Glucocorticosteroid. Inhibits the function of leukocytes and tissue macrophages. The migration of leukocytes in the area of inflammation. Impair the capacity of macrophages to phagocytosis and the formation of interleukin-1. Contributes to the stabilization of lysosomal membranes, thereby reducing the concentration of proteolytic enzymes in the field of inflammation. Decreases capillary permeability due to histamine release. Inhibits activity of fibroblasts and collagen formation. Inhibits the activity of phospholipase A2 which leads to suppression of the synthesis of prostaglandins and leukotrienes. Suppresses the release of COX (mainly COX-2), which also helps to reduce production of prostaglandins. Reduces the number of circulating lymphocytes (T-and B-cells), monocytes, eosinophils and basophils as a result of their displacement from the vascular bed to lymphoid tissue, suppresses the formation of antibodies. hydrocortisone suppresses the release of pituitary ACTH and beta-lipotropin but does not reduce the level of circulating beta-endorphin. Inhibits the secretion of TSH and FSH. With direct application of the receptacles has a vasoconstrictor effect. hydrocortisone has a pronounced dose-dependent effect on the metabolism of carbohydrates, proteins and fats. Stimulates gluconeogenesis, promotes the capture of amino acids by the liver and kidneys and increases the activity of enzymes of gluconeogenesis. In the liver of hydrocortisone increases glycogen deposition by stimulating the activity of glycogen synthase and synthesis of glucose from the products of protein metabolism. Increased blood glucose activates the secretion of insulin. hydrocortisone suppresses the seizure of glucose by fat cells that leads to activation of lipolysis. However, due to increased secretion of insulin is stimulation of lipogenesis which leads to the accumulation of fat. This medication has catabolic effect in lymphoid and connective tissue, muscle, adipose tissue, skin, bone tissue; to a lesser extent than the mineralocorticoid affects the processes of water and electrolyte metabolism: promotes the excretion of potassium ions and calcium, delay in the body of sodium and water. Osteoporosis and Itsenko-Cushing\'s syndrome are the main factors limiting the long-term therapy of GCS. As a result of catabolic actions may suppress growth in children. In high doses hydrocortisone may increase the excitability of brain tissue and contributes to lowering the threshold of convulsive readiness. It stimulates the excessive production of hydrochloric acid and pepsin in the stomach that promotes the development of peptic ulcers. For systems use therapeutic activity of hydrocortisone caused by anti-inflammatory, anti-allergic, immunosuppressive and antiproliferative action. When external and local application the therapeutic activity of hydrocortisone is due to anti-inflammatory, anti-allergic and antiexudative (due to vasoconstrictor effect) action. For anti-inflammatory activity this medicine is 4 times weaker than prednisolone, at mineralocorticoid activity is superior to other GCS. Indications: dry skin, hyperkeratosis Indications: skin disorders Indications: infected skin disorders indicated for the relief of the inflammatory manifestations of corticosteroid responsive dematoses when complicated by secondary infection, Caused by organisms susceptible to gentamicin Clioquinol is an anti-infective agent which has both antibacterial and anticandidal activity. Calmurid Alpes Chemie is a glucocorticosteroid. Calmurid Alpes Chemie inhibits the function of leukocytes and tissue macrophages. The migration of leukocytes in the area of inflammation. Impair the capacity of macrophages to phagocytosis and the formation of interleukin-1. Contributes to the stabilization of lysosomal membranes, thereby reducing the concentration of proteolytic enzymes in the field of inflammation. Decreases capillary permeability due to histamine release. Inhibits activity of fibroblasts and collagen formation. (Systemic use):Endocrine disordes ( primary or secondary adrenocortical insufficiency ).shock unresponsive to conVentional therapy if adrenocortical insufficiency exists or is suspected, Rheumati c disorders ( post – traumatic osteoarthritis, synovitis of osteoarthritis, Rheumatoid arthritis, Acute and subacute bursitis. Epicondylitis, Acute nonspecific tenosynovitis, Acute gouty arthritis, Psoriatic arthritis Collagen diseases ( systemic lupus erythematosus, Acute rheumatic Carditis, systemic dermatomysitis ), Dermatologic disease ( Pemphigus, Severe erythma multiforme, Exfoliative dermatitis, Severe psoriasis ) ophthalmic diseases, ( in allergic and inflammatory processes involving the eye, such as : Herpes Zoster ophthalmicus, Iritis, iridocy clitis, chorioretinitis, Keratitis ), Gastrointestinal diseases ( ulcerative colitis systemic theropy ) Respiratory diseases ( symptomatic sarcoidosis, Berylliosis ) Hematologic disorders ( Acquired [ autoimmune ] hemolytic anemia ) Neoplastic disease. (Topical use):variety of skin conditions (e.g., insect bites, poison oak/ivy, eczema, dermatitis, allergies, rash, itching of the outer female genitals, anal itching). Hydrocortisone reduces the swelling, itching, and redness that can occur in these types of conditions. This medication is a mild corticosteroid.
更新日期: 2014-03-11
附件:
201441021274714.pdf    

 
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